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Concurrently, diminished GSH synthesis in OA chondrocytes impairs cellular antioxidant capacity, sensitizing cells to copper toxicity

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Furthermore, exposure to foreign toxicants such as those present in CS or HTP mainstream can trigger the secretion of various immune mediators, including cytokines from the pulmonary compartment, inducing a condition of systemic inflammation, thereby affecting the circulatory system and contributing to an increased risk of atherosclerosis [39]